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Correction: ASPM-associated stem cell proliferation is involved in malignant progression of gliomas and constitutes an attractive therapeutic target.

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HAL Id: inserm-00663664

https://www.hal.inserm.fr/inserm-00663664

Submitted on 27 Jan 2012

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Correction: ASPM-associated stem cell proliferation is

involved in malignant progression of gliomas and

constitutes an attractive therapeutic target.

Sandra-Nadia Bikeye, Carole Colin, Yannick Marie, Raphaël Vampouille,

Philippe Ravassard, Audrey Rousseau, Blandine Boisselier, Ahmed Idbaih,

Charles Calvo, Pascal Leuraud, et al.

To cite this version:

Sandra-Nadia Bikeye, Carole Colin, Yannick Marie, Raphaël Vampouille, Philippe Ravassard, et al..

Correction: ASPM-associated stem cell proliferation is involved in malignant progression of gliomas

and constitutes an attractive therapeutic target.. Cancer Cell International, BioMed Central, 2011,

11 (1), pp.10. �10.1186/1475-2867-11-10�. �inserm-00663664�

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C O R R E C T I O N

Open Access

Correction: ASPM-associated stem cell

proliferation is involved in malignant

progression of gliomas and constitutes

an attractive therapeutic target

Sandra-Nadia Ngwabyt Bikeye

1

, Carole Colin

2

, Yannick Marie

1

, Raphaël Vampouille

4

, Philippe Ravassard

4

,

Audrey Rousseau

5

, Blandine Boisselier

1

, Ahmed Idbaih

1,3

, Charles Félix Calvo

1

, Pascal Leuraud

6

, Myriam Lassalle

6

,

Soufiane El Hallani

1

, Jean-Yves Delattre

1,3

and Marc Sanson

1,3*

Correction

In the original article [1], the additional file two: figure S.

Two (additional file 1 here) contained two identical

graphs. We present here the corrected figure S2 and the

corresponding legend:

Additional material

Additional file 1: Tumor spheroid characterization. Genomic stability was examined with CGHa analysis (left = DNA profile from the initial tumor; right = DNA profile from gliomasphere at passage p28). The chromosomes are indicated on the × axis and copy number is on y axis. Yellow indicates the normal genomic copy number, while green indicates a loss and red indicates a gain in copy number. Although some differences are observed, the comparison of both profiles shows that overall genomic profile is quite well preserved (except for an amplicon on chromosome 8 present only in the gliomasphere).

Author details

1

UMR975 INSERM-UPMC, GH Pitié- Salpêtrière, Paris, France.2UMR911-CRO2, Faculté de Médecine Timone, Université de la Méditerranée, Marseille, France.3Service de Neurologie Mazarin, GH Pitié- Salpêtrière, APHP, Paris,

France.4Biotechnology & Biotherapy laboratory, Centre de Recherche de

l’Institut du Cerveau et de la Moelle, CNRS UMR9225, INSERM UMRS 975 Université Pierre et Marie Curie, Hôpital de la Salpêtrière, Paris, France.

5Service de Neuropathologie Raymond Escourolle, GH Pitié- Salpêtrière, Paris,

France.6XenTech, Evry, France.

Received: 8 April 2011 Accepted: 15 April 2011 Published: 15 April 2011 Reference

1. Bikeye Ngwabyt Sandra-Nadia, Colin Carole, Marie Yannick, Vampouille Raphaël, Ravassard Philippe, Rousseau Audrey,

Boisselier Blandine, Idbaih Ahmed, Calvo Félix Charles, Leuraud Pascal,

Lassalle Myriam, El Hallani Soufiane, Delattre Jean-Yves, Sanson Marc: ASPM-associated stem cell proliferation is involved in malignant progression of gliomas and constitutes an attractive therapeutic target. Cancer Cell International 2010, 10:1.

doi:10.1186/1475-2867-11-10

Cite this article as: Bikeye et al.: Correction: ASPM-associated stem cell proliferation is involved in malignant progression of gliomas and constitutes an attractive therapeutic target.Cancer Cell International 2011 11:10.

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Submit your manuscript at www.biomedcentral.com/submit * Correspondence: marc.sanson@psl.aphp.fr

1UMR975 INSERM-UPMC, GH Pitié- Salpêtrière, Paris, France

Full list of author information is available at the end of the article Bikeyeet al. Cancer Cell International 2011, 11:10

http://www.cancerci.com/content/11/1/10

© 2011 Bikeye et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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