• Aucun résultat trouvé

Fungal secondary metabolites as effectors of pathogenicity:role in the complex interplay between rice and Magnaporthe grisea

N/A
N/A
Protected

Academic year: 2021

Partager "Fungal secondary metabolites as effectors of pathogenicity:role in the complex interplay between rice and Magnaporthe grisea"

Copied!
6
0
0

Texte intégral

(1)

HAL Id: hal-02816584

https://hal.inrae.fr/hal-02816584

Submitted on 6 Jun 2020

HAL is a multi-disciplinary open access

archive for the deposit and dissemination of sci-entific research documents, whether they are pub-lished or not. The documents may come from teaching and research institutions in France or abroad, or from public or private research centers.

L’archive ouverte pluridisciplinaire HAL, est destinée au dépôt et à la diffusion de documents scientifiques de niveau recherche, publiés ou non, émanant des établissements d’enseignement et de recherche français ou étrangers, des laboratoires publics ou privés.

Fungal secondary metabolites as effectors of

pathogenicity:role in the complex interplay between rice

and Magnaporthe grisea

Jérôme Collemare, Rahadati Abdou, Marie-José Gagey, Zhongshu Song,

Walid Bakeer, Russell Cox, Elsa Ballini, Didier Tharreau, Marc-Henri Lebrun

To cite this version:

Jérôme Collemare, Rahadati Abdou, Marie-José Gagey, Zhongshu Song, Walid Bakeer, et al.. Fungal secondary metabolites as effectors of pathogenicity:role in the complex interplay between rice and Mag-naporthe grisea. ECFG10 : 10. European conference on fungal genetics, Mar 2010, Noordwijkerhout, Netherlands. pp.41. �hal-02816584�

(2)
(3)
(4)
(5)
(6)

Références

Documents relatifs

Studying co- nidial pigmentation in Aspergillus nidulans (A. nidulans), they showed that wA gene encodes a polyketide synthase respon- sible for the formation of the linear monomer

Bastien Bissaro, Sayo Kodama, Mireille Haon, David Ribeaucourt, Mickael Lafond, Harry Brumer, Richard O’Connell, Yasuyuki Kubo, Jean-Guy Berrin.. To cite

In addition, based on phenotypic characterization of interactions and population genetics of the pathogen, we will show evidences of a longer coevolution process that leaded

To confirm a role of these WAKs in rice blast resistance, Knock-Out and transgenic rice lines with constitutive expression of a subset of WAKs were produced. Kinase

In the absence of recombination, and because of short distance migrations, selection for an increased virulence spectrum to attack upland cultivars led to differentiation

The diversity and structure of populations of Magnaporthe grisea, the main fungal pathogen of rice, was described in many countries during the last 20 years.. The expected

ACE1 / Pi33 : our model system for specific interactions Pi33 pi33 ACE1 ace1 Plant Pathogen. Fudal

Chlorimuronethyl Resistance Selectable Marker Unsuited for the transformation of rice blast fungus (Magnaporthe grisea).. CHANG Qing † , YANG Jing † , LIU Lin, SU Yuan, LI Jinbin,