• Aucun résultat trouvé

Acute Induction of Translocon-Mediated Ca2+ Leak Protects Cardiomyocytes Against Ischemia/Reperfusion Injury

N/A
N/A
Protected

Academic year: 2021

Partager "Acute Induction of Translocon-Mediated Ca2+ Leak Protects Cardiomyocytes Against Ischemia/Reperfusion Injury"

Copied!
18
0
0

Texte intégral

Loading

Figure

Figure 2. The reticular puromycin‐mediated Ca 2+  stock release was independent of the one stimulated  by  caffeine  and  did  not  affect  excitation–contraction  (E‐C)  coupling.  (A,D)  Time  traces  showing  cytosolic  Ca 2+   concentration  assessed 

Références

Documents relatifs

The results of the current study indicated that in the case of ARVMs that underwent sI/R injury, the administration of exogenous rhSLPI, prior to or at the onset of sI significantly

Contribution of myeloid HO-1 to the modulation of renal ischemia-reperfusion injury Effect of myeloid HO-1 induction with hemin as a preemptive treatment strategy against

From here, several injury cascades are activated, including activation of cell death programs like apoptosis and (regulated) necrosis, endothelial dysfunction implicating

We have demonstrated, both in vitro and in vivo models of ischemic stress as it occurs during cold ischemia, that kidney epithelial cells subjected to glucose deprivation

A comparison of hypothermic machine perfusion versus static cold storage in an experimental model of renal ischemia reperfusion injury. Renoprotective effect of pulsatile

We further demonstrate in healthy animals a reduction of QTc interval mediated by NO/cGMP pathway leading to the shortening of ventricular cardiomyocytes action potential duration

We further demonstrate in healthy animals a reduction of QTc interval mediated by NO/cGMP pathway leading to the shortening of ventricular cardiomyocytes action potential duration

Resveratrol improves mitochondrial energetic pathway and endothelial function in type 2 diabetic female rat heart submitted to ischemia-reperfusion injuryN.