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Hepatitis C virus drives increased type I interferon-associated impairments associated with fibrosis severity in antiretroviral treatment-treated HIV-1-hepatitis C virus-coinfected individuals

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Figure

Table 1. Study patients’ characteristics. Groups HIV-1-monoinfectedindividuals,n¼14 HIV-1–HCV-coinfectedindividuals,n¼15 Healthy controls,n¼13 P value General characteristics
Fig. 1. Comparison of the plasma levels of inflammatory markers between hepatitis C virus–HIV-1 coinfected, HIV-1- HIV-1-monoinfected treated individuals and healthy controls
Fig. 2. Persistent activation of IFN a signaling in plasmacytoid dendritic cells (pDCs) in HIV-1-infected individuals despite suppressive antiretroviral treatment, irrespective of hepatitis C virus (HCV) status
Fig. 3. Transcriptomic analysis of plasmacytoid dendritic cells (pDCs) from hepatitis C virus (HCV)–HIV-1-coinfected individuals with minimal-to-moderate fibrosis, severe fibrosis, HIV-1-monoinfected individuals and healthy donors
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