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Inhibition of neuronal FLT3 receptor tyrosine kinase alleviates peripheral neuropathic pain in mice

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Academic year: 2021

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Fig. 1 In fi ltration of CCI-injured nerve by CD45/FL-positive cells and effects of intrathecal FL injections on pain sensitivity and PNP-related biomarkers
Fig. 2 FL potentiates TRP function in vitro and in vivo. a Traces of [Ca 2+ ] i responses to repeated bath applications of capsaicin (Cap, 2.5 µ M), a selective TRPV1 activator, or capsaicin combined with FL (0.56 nM) on [Ca 2+ ] i levels measured by real-
Fig. 3 FLT3 is critical for the development of pain hypersensitivity. a – g Flt3 -knockout ( Flt3 KO ), compared to wild-type (WT) animals, showed marked reductions of hypersensitivity to mechanical nociceptive stimuli as measured by the von Frey test (a),
Fig. 4 FLT3 is critical for upregulation of markers associated with peripheral nerve injury
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