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Aberrant Glycosylation Promotes Lung Cancer Metastasis through Adhesion to Galectins in the Metastatic Niche

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Academic year: 2021

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Figure 1. Mice bearing tumors have accumulation of the adhesion ligand, galectin-3, in the early  metastatic niche
Figure 2. Tumor-derived soluble factors induce the mobilization of galectin-3 +  myeloid cells into  peripheral circulation early in tumorigenesis
Figure 3. Secretion of IL-6 by tumors induces the rapid mobilization of CD11b + galectin-3 +  leukocytes
Figure 4. Elevated Thomsen-Friedenreich Antigen presentation promotes increased galectin-3  adhesion in metastatic populations
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