• Aucun résultat trouvé

Peripheral Blood Mononuclear Cell Metabolism Acutely Adapted to Postprandial Transition and Mainly Reflected Metabolic Adipose Tissue Adaptations to a High-Fat Diet in Minipigs

N/A
N/A
Protected

Academic year: 2021

Partager "Peripheral Blood Mononuclear Cell Metabolism Acutely Adapted to Postprandial Transition and Mainly Reflected Metabolic Adipose Tissue Adaptations to a High-Fat Diet in Minipigs"

Copied!
21
0
0

Texte intégral

Loading

Figure

Figure 1. Plasma parameters in Yucatan mini-pigs fed either a control or a high fat–high sucrose  (HFHS) test meals
Figure 2. Hexokinase (HK), pyruvate kinase (PK), lactate dehydrogenase (LDH), glucose 6-phosphate  dehydrogenase (G6PD), malate dehydrogenase (MDH), branched chain amino-acid transaminase  (BCAT), glutaminase P-dependent, aspartate aminotransferase (AspAT)
Figure 3. Phosphorylation levels of Akt Ser 473  and eEF2-α Thr 56  in PBMCs and S6 Ser 235/236  from  Yucatan mini-pigs fed either a control or a high fat–high sucrose (HFHS) test meals
Table 1. Plasma fasting metabolites in Yucatan minipigs fed a high fat–high sucrose diet (HFHS) over 2 months.
+6

Références

Documents relatifs

TABLE 5: Growth parameters and Nhe production determined from uncontrolled batch cultures of ldhA, pfo, ldhB and ldhC mutants and their parent strain F4430/73 (wt).

Assessment of glucose-6-phosphate dehydrogenase activity using CareStart G6PD rapid diagnostic test and associated genetic variants in Plasmodium vivax malaria endemic setting

Their results revealed that neither C149S nor C149S/C281S mutants aggregated, proposing that the active site cysteine of GAPDH plays an essential role in amyloid binding,

Molecular analysis of glucose-6-phosphate dehydrogen- ase deficiency in this study conducted in Jeddah revealed a higher prevalence of G6PD Mediterranean (89.1%) and Aures

To assess inter- and intra-laboratory variability in G6PD spectrophotometry, data from the measurement of manufacturer-provided quality control samples were used to quantify

Since in two sites (Vietnam and Laos) only phenotypically deficient and intermediate samples were genotyped (and the propor- tion of heterozygous women with normal G6PD pheno-

Furthermore, SAR analysis of EA analogues supported by docking models provides clues towards the determinants for the selective inhibition of the pathogen enzyme by steroids: (i)

We coupled these results with compu- tational biology analyses along with subcellular localization experiments to propose two mitochondrial kinase enzymes (T. gondii PDK [TgPDK] and