Hepatitis E Virus Lifecycle and Identification of 3 Forms of the ORF2 Capsid Protein

In patient 2, TT-speci fic CD4 + T-cell responses were negative but turned positive after treatment, most likely as an effect of immune reconstitution by rhIL-7 (Figure 1 D)..

We transfected cells with expression vectors for protein VI-wt, VI-M1 and VI-delta54 (Figure 9A) and HCMV tegument protein pp71 and HPV small capsid protein L2 (Figure 9B) followed

Vesicular stomatitis virus replicon expressing the VP2 outer capsid protein of bluetongue virus serotype 8 induces complete protection of sheep against challenge infection... R E S E

protein showed similar reaction values to the purified scFv in the ELISA test, and the results of ELISA analysis using IgY-scFv toward CPV clinical samples were consistent

By combining the highly replicative and cell culture-selected gt3 p6 strain 19 and a highly transfectable subclone of PLC/PRF/5 cells (PLC3 cells), we recently developed an

viral RNA load, RBV therapy caused a reduction in the level of ORF2 in both the stool and the plasma 192. of the treated mice

In order to identify the major MCPyV VP1 conformational epitopes, we investigated the re- activity of wild-type and four VP1 protein insertional mutants against a panel of

To show that the Daxx displacement from PML-NBs in the very early infection phase was caused by protein VI, we analyzed Daxx dissociation from PML- NB also in VI-wt and