Ethyl-eicosapentaenoate modulates changes in neurochemistry and brain lipids induced by parkinsonian neurotoxin 1-methyl-4-phenylpyridinium in mouse brain slices

Dietary intake of omega-3 fatty acids therefore clearly appears to modulate brain glucose uptake, a mechanism by which omega-3 fatty acid status may influence cognitive function

12-HETE is produced by the 12/15 LOX enzyme and conver- sion of AA by 12-HETE is enhanced in n-3 PUFA deficient microglia in the context of brain injury, autoimmune and

Ca 2+ currents associated with backpropagating action potentials in CA1 hippocampal pyramidal neurons and in olfactory bulb (OB) mitral cells (a) On the left, fluorescence

5fC: 5-Formylcytosine; 5hmC: 5-Hydroxymethylcytosine; 5mC: 5- Methylcytosine; AD: Alzheimer ’s disease; AgeCoDe: Study on Ageing, Cognition and Dementia in Primary Care Patients

examine interrelationships between disease duration, fear of dyspnea/ physical activity, and GMV in brain areas where patients demonstrated reduced GMV (using the gray scale

Supplemental Figure 4: Kinetics of T-cell brain infiltration and glial cell activation in the SNpc of MPTP-intoxicated mice. Representative photomicrographs of mesencephalic

The immunohistochemical detection of influenza virus showed that viral infection in the brain occurred as early as 1 dpi and that the virus was detected until 5 dpi (Table 2).

mRNA and/or protein expression of inflammasome components and substrates NLRP1, NLRP3, NLRC4, ASC (Pycard), Aim-2, Caspase-1, Caspase-4, IL-1α, IL-1β, IL-18 and HMGB1, were studied