Progenitor cell expansion and impaired hepatocyte regeneration in explanted livers from alcoholic hepatitis

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For the prediction of liver ‐ related death the number of events at 10 years was too small (n = 7) for any conclusion. Therefore, the primary aim was to assess the prognostic value

Results showed that in all cases the vineyard soil was a large reservoir of yeast, and the main differences were obtained between the non-degraded area and the manure restored

In order to gain insight into the main drivers of gene expression that could result in the devel- opment of hepatocellular failure in AH, we analyzed the predicted activity

19 It has been shown that the IL-33/ST2 pathway attenuates sepsis in the spe- ci fi c setting of infection without liver impairment by enhancing neutrophil in fl ux to the site

In sum, the above data suggest that GPS2 repression of PPARα target genes in hepatocytes involves direct interactions of PPARα with GPS2 and NCOR, which serves as the primary

Patients: Ninety-three patients with compensated or decompensated cirrhosis (CLF group) but stable liver function and 30 patients with cirrhosis and hospitalized for the management

Supplementary Table 1: Differences in intestinal microbiota at the genus level between patients with alcoholic cirrhosis, with (Cir_sAH ) and without severe alcoholic