NADPH oxidases in Parkinson’s disease: a systematic review

BACKGROUND: Our preceding study revealed that NOX4 expression is regulated in an anti-adipogenic fashion and inversely related to adipocyte differentiation in white and brown

In microglia, mGlu 3 and mGlu 5 are the dominant metabotropic glutamate receptors expressed (Pocock and Kettenmann, 2007; Berger et al., 2012), and of these, the mGlu 5 receptor

Using the patch- clamp technique, one can thus measure several parameters that directly or indirectly reflect the activity of the NADPH oxidase, namely: (i) the plasma membrane

Although no suitable antibody is available for NOX2 im- munohistochemistry in mice, the following indirect evidence indicate microglial expression of NOX2: (i) concomitant increase

The optimal density of Ra 2 microglial cells (90'000/well) was selected (Fig. The effects of Ra 2 microglial cell density in neuroblastoma- microglial co-culture system. After 48

(C-D) Quantitative real-time PCR was performed also on mRNA from the spinal cord of two additional mouse models of ALS: (C) Increase in mRNA for NOX2 was found in the spinal cord

However, in a model of autoimmune multiple sclerosis, in vivo data on the role of the phagocyte NADPH oxidase system in myelin oligodendrocyte glycoprotein (MOG)- induced

The optimal density of Ra 2 microglial cells (90'000/well) was selected (Fig. The effects of Ra 2 microglial cell density in neuroblastoma- microglial co-culture system. After 48