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CYP2E1 genotype and isoniazid-induced hepatotoxicity in patients treated for latent tuberculosis

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Academic year: 2021

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Figure

Fig. 1 Pathways of isoniazid metabolism. Scheme illustrating the metabolism of isoniazid through multiple pathways
Table 1 Incidence a of isoniazid-induced hepatitis (INH-H) and isoniazid-induced elevated liver enzymes (INH-ELE) as a function of sex and age
Table 3 Susceptibility of INH-H and INH-ELE in each genotype group
Table 5 Prognostic value a of the CYP2E1 polymorphism for determining isoniazid-induced hepatotoxicity

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