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Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone Axis

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Academic year: 2021

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Fig 5. The H3.3K9me3 chromatin mark is present on vDCP NBs-associated latent/quiescent HSV-1 genomes
Fig 6. Individual inactivation of DAXX, ATRX, HIRA, or UBN1 significantly affects the formation of vDCP NBs, but only mildly affects the association of H3.3 with the latent/quiescent HSV-1 genome
Fig 8. Absence of PML decreases the co-localization of DAXX and ATRX but not HIRA and UBN1 with latent/quiescent HSV-1 genomes
Table 1. Characteristics of the shRNA-expressing plasmids.
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