• Aucun résultat trouvé

Selenoproteins and Protection against Oxidative Stress: Selenoprotein N as a Novel Player at the Crossroads of Redox Signaling and Calcium Homeostasis

N/A
N/A
Protected

Academic year: 2021

Partager "Selenoproteins and Protection against Oxidative Stress: Selenoprotein N as a Novel Player at the Crossroads of Redox Signaling and Calcium Homeostasis"

Copied!
13
0
0

Texte intégral

Loading

Figure

FIG. 1. Schematic diagram depicting the different sites of ROS and NO generation by cells (italic font) and the main antioxidant enzymes present in a majority of human cell types (shadowed)
FIG. 3. Putative role of SelN in redox homeostasis of human muscle cells.
FIG. 4. Mechanisms of action and pathophysiological targets of N-acetyl cysteine (NAC) in SEPN1-related  my-opathy

Références

Documents relatifs

We describe the particle motion in a vertical tube of the same diameter as those used in a solar receiver, investigated through the use of the Positron Emission Particle Tracking

Particle in cell simulations were carried out with a plasma bounded by two absorbing walls and a magnetic field applied parallel to them. Both the sheath extent and the potential

In addition, this voluntary wheel running protocol decreased brain disorders (BBB leakage and macrophage accumulation) and aortic plaque size, and increased aortic

To induce basidiomata production, we with- drew water for 15-20 days (water stress). Six phases of fungal development on the cookies were studied: 1) white phase, corresponding to

The oxidative stress linked to inflammation plays an important role in (i) endothelial dysfunction, with reduced nitric oxide (NO) bioavailability, (ii)

gene expression and modulates fermentation product formation and oxidative stress

On a dry mass basis sun exposed leaves of seedlings and adult canopy trees displayed significantly higher amounts of total ascorbate and a-tocopherol (figure 1) and lower

Regarding the expression of anti-inflammatory genes, AO supplementation had a specific effect, by inducing both IL-4 and IL-10 mRNA levels in AO + LPS mice, while OO supplementation