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Caractérisation de l'interaction de l'auto-antigène ADN topoisomérase I avec les fibroblastes dans la sclérose systémique

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Academic year: 2021

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Figure

Figure 1.  Manifestations cliniques associées à la sclérose systémique.
Tableau I.  Caractéristiques des modèles animaux pour l’étude de la sclérose   systémique
Figure 2. Modèle pathophysiologique de la sclérose systémique. Agent étiologique Susceptibilité géniqueFacteurs environnementaux 2-Recrutement de cellules mononuclééesActivation des macrophages et des LT 6-Différenciation en myofibroblastes
Figure 3. Patrons d’immunofluorescence indirecte sur cellules HEp-2 des auto-anticorps
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