L’adhérence de microorganismes aux cellules intestinales induit une réponse immunitaire de type Th17

staining, we validated that SIRT1 agonists reduced IL-17A expression in human Th17 cells (Figure 5G).. The SIRT1 antagonist EX-527 restored IL-17A expression

The DSC data were then used to determine the contribution of the secondary crys- tallization mechanism, considering that primary crystallization is related to the

In-plane micro-photoluminescence (µ-PL) and micro-reflectivity measurements have been performed at room temperature by optical excitation perpendicular to the surface of

CD4 + cells were isolated from PBMC by magnetic sorting, expanded in a culture medium without serum in the presence of polarizing cytokines and TCR-stimulation, activated,

The percentages of surface-labeled IL-17A-secreting cells were quite similar to the percentages of intracellular-labeled IL- 17A-producing cells of the same cultures, and the

(A) Left, Overview of the cryo-EM reconstruction of Arp2/3-C1B-C5L with the docked model in the density, viewed and coloured as originally presented by (Robinson et al., 2001): Arp2:

Par les expériences en chambre «transwell», nous avons aussi pu émettre l'hypothèse que le contact cellulaire agit en synergie avec la production de cytokines induite par